More from @DrJackKruse

Oct 23

1. Today’s PSA:

2. Hi Jack,

Wondering if you’d be able to break down what happened after I took Venlafaxine (Effexor) a few years ago and if there’s any specific things needed to be done to hopefully repair any damage.

After what I believe was a spontaneous CSF leak that happened one night and started all my chronic issues I was diagnosed with vestibular migraine around 3 months later.

Venlafaxine 75mg was indicated for this so I was on it for around 12 months (give or take the 3 months wean I did).

Within 3 weeks my emotions went numb, I lost my libido, erections and ability to pretty much feel love or any strong emotion. There was a very definite and obvious “change” but I believe I still had the ability to focus and enjoy hobbies etc.

After deciding to taper off, I would get brain zaps and all the other jazz that comes with it. After fully tapering, I was then introduced to anhedonia. Unable to feel or enjoy anything, unable to focus on anything for too long without getting the irritable and feeling like I need to do something else etc. Essentially feel there’s been a permanent change since starting this.

This has improved slightly but not even close to where I’d like to be. Add all the daily brain fog, memory lapses etc and my brain is running on empty.

Does this need anything specific additional to the core light, water & magnetism?

Listening to this months webinar (October 2025) you mentioned accutane extending recovery from 6 months up to 5 years. This prompted me to ask this question and about my accutane use in the future as this is a huge jump in recovery time and shows how powerful these drugs are.

3. Reasons Why Venlafaxine Could Cause Issues, Based on My Thesis

My thesis posits that life’s core “code” resides in conserved correlations of phase, spin, and topology, geometric invariants that maintain coherence beyond mere voltage gradients. Man-made electromagnetic (EM) pollution disrupts this coherence, leading to health risks like aging, cancer, and failed regeneration. Voltage is merely a “courier,” while true memory and form emerge from photonic organization of spin and topological symmetry.

Venlafaxine, as a synthetic molecule introduced into this already polluted EM environment, would act to exacerbate disruptions by interfering with biological EM phenomena. Below, I list hypothesized reasons grounded in its structure, spectra, pharmacology, and potential EM interactions.

These are hypothestical extensions based on my published framework, drawing from known drug effects and bio-EM principles (e.g., biophotons, ion flows, and field coherence), while acknowledging that direct studies on venlafaxine-EM-biology intersections are limited because BigHarma isn’t going to dump on its own product with truthful decentralized science.

Read 20 tweets

Oct 18

1. The terms anaplerosis and cataplerosis describe reciprocal and correlative reactions involved in the function of the TCA/urea cycle. The enzymatic steps in these processes have long been known, but the overall concept of a linkage between anaplerosis and cataplerosis should be underscored because the balance between these two processes controls the entry and exit of TCA cycle anions.

Anaplerotic and cataplerotic reactions are involved in the ultimate disposal of all metabolic intermediates. The metabolic role of anaplerosis and cataplerosis in amino acid metabolism varies with the light environment, specific organs and is dependent on the nutritional/metabolic status of the individual.

If the AM sunrise is seen by the eye, skin, and sensed by the gut via the skin clocks, during feeding, the intestine is an important site of catabolism of enterally derived amino acids, whereas in the starved state amino acid catabolism occurs primarily in the kidney, liver, and muscle. The light environment is critical in how anaplerosis and cataplerosis operate in people.

Every tissue differs in how it uses anaplerosis and cataplerosis.This implies the regulation of anaplerosis and cataplerosis is very dependant on deuterium kinetics in the matrix from normal or abnormal metabolic and physiologic states.

Methionine: Propionyl-CoA forms as a catabolite of methionine, threonine, and the branched-chain amino acids. β-Oxidation of fatty acids with an odd number of carbon atoms yields propionyl-CoA. The oxidation of the side chain of cholesterol also yields propionyl-CoA. Thus, propionyl-CoA is derived from the catabolism of lipids and proteins.Propionyl-CoA is converted to succinyl-CoA, which is oxidized or converted to glucose by way of oxaloacetate and pyruvate. Succinyl-CoA may also form δ-aminolevulinate, a precursor of porphyrin biosynthesis.

This is critical in oncogenesis because cancer cells need brisk ECT which means that cancer cells can use methionine to usurp oxygen using methionine to increases both angiogenesis and hemoglobin production to make sure that oxygen delivery is brisk and apoptosis stays inhibited PROVIDED VDR/D3 and/or UVA are absent to slow ECT flow.

NO is used to slow ATPase as a braking mechanism when oxygen is a toxin. This is a remnant from our GOE evolution before heme proteins and melanin were innovated to protect us as a firewall from oxygen. NO is liberated by UVA light and NIR.

Update your biochemical models because they are all broken.

2. Anaplerosis and Cataplerosis: They are Light-Dependent Gatekeepers of TCA/Urea Cycle Dynamics and Metabolic Fate

From first principles, metabolism is fundamentally an energy and information flow process, governed by thermodynamic gradients where sunlight provides the primary low-entropy input to drive organization and efficiency.

The tricarboxylic acid (TCA) cycle, also known as the Krebs or citric acid cycle, serves as life’s central hub for oxidizing nutrients to generate ATP, but it doesn’t operate in isolation.

Anaplerosis (replenishment of TCA intermediates like oxaloacetate or succinyl-CoA) and cataplerosis (removal of these intermediates for biosynthesis or disposal) act as reciprocal OPTICAL valves, ensuring the cycle’s continuity while adapting to cellular demands

3. Ultimately, these processes dictate the disposal of all metabolic intermediates, linking carbon, nitrogen, and sulfur flows across pathways like amino acid catabolism, fatty acid oxidation, and urea cycle.This balance controls the entry and exit of TCA anions (e.g., citrate, malate), preventing depletion during high biosynthetic pulls (e.g., gluconeogenesis, lipid synthesis) or overflow during nutrient excess.

Ultimately, these processes dictate the disposal of all metabolic intermediates, linking carbon, nitrogen, and sulfur flows across pathways like amino acid catabolism, fatty acid oxidation, and urea cycle.

Read 19 tweets

Oct 15

There is only one kind of shock worse than the totally unexpected outcome: the expected for which one has refused to prepare. This is the new jouney for those who complied with tyranny and joined the experimental group the last 5 years.

The expected always happens and this can be a problem too. Sometimes the most scenic roads in life are the detours you didn’t mean to take. This is true for the control group.

The results of traveling the road less traveled surprises us. So, would you really like to know your future?

If your answer is yes, think again. Not knowing is the greatest life motivator for thriving for those of us who rejected compliance. So enjoy, endure, survive each moment as it comes to you in its proper sequence – a surprise. Embrace the chaos and the suck for a change, but never comply with tyranny.

2. People who complain they have no time or wish they could control how they spend their time to do what they should are stuck in a low dopamine state because of the light they live under. Few of them are aware of the implications.

Here is how it works: Know how to get the most results in the least amount of time. That’s the ultimate aim of productivity skills. Savages know we all have the same amount of time in a day so when somebody tells you they do not have time to do something they have a made choice with their time to do something else. In the age of information, ignorance of the wisdom of reality and nature is a choice.

3. With that said how come equatorial Africans never seem to get cancers on the equator when they make the daily choice to be in the strongest sun on the planet possible, but people who have no time because they are working under fake light and nnEMF seem to get all denovo or jab induced cancers, and especially the worse types?

Read 5 tweets

Oct 15

He said, “the same command becomes a metabolic loan the body can’t repay.”

This is why evolution put the VDR recpetor on the IMM. When you have UV and IRA/NIR you can repay the loan. This means the SUN is TINA. Why? It slows the charge on the IMM.

It should not make sense why I do use affliate codes for red lights.

They are a half truth to those with electrical resistance problems in the IMM.

You always need purple and red to pay the loan back. Purple and red together renovate the poor engines to restore the default state.

https://x.com/DrJackKruse/status/1978432850266304689

2. The U.S. has the highest first-day infant mortality out of industrialized world About 11,300 newborns die within 24 hours of their birth in the U.S. each year, 50 percent more first-day deaths than all other industrialized countries combined. This is that awkward moment when the statistics confirm that the experts are wrong about something, and the children are dying because of it. The problem in the U.S. is that many of the babies born here are premature.

cbsnews.com/news/us-has-hi…

What are the reasons that babies are born prematurely? Risk Factors for Premature Birth are all correlated with artificial light melanopsin damage. Can you imagine that? Many risk factors can be reduced or eliminated altogether. Talk to your doctor or midwife about your individual risk factors and what you can do to diminish them.

Risk factors include:

Prior premature birth. jaundice requiring blue light therapy Multiple pregnancies. Uterine or cervical problems. Chronic high blood pressure. Diabetes. Smoking. Jab compliance. Age. Lack of UV exposure prenatally. Poor nutrition due to missing DHA. Untreated infection.

3. What do bears and humans have in common? Both are carnivore mammals who have adapted to high latitude living over their evolutionary history. Bear testosterone levels peak in the Spring and early Summer in high latitudes when UV light and IRA/NIR light return in unison to their environments.

Why does their hormone panel change?

Terrestrial solar light determines mating and fertility in mammals at all laltitudes but it most obviously seen at high latitudes where UV is not stable yearly.

What happens when humans bombard themselves with fake light they make?

You get an infertility epidemic in humans! = FACTOR Y on display.

This makes the OB/GYN’s happy and rich. It also creates a lot of misgendering, beta, and mental illness. It also creates a lot of suboptimal children with developmental issues. We should never force nature into doing something she says should not occur.......unless we are ready for the collateral damages. academic.oup.com/biolreprod/art…

Read 4 tweets

Oct 14

1. How will the transhumanist technologists use AI in the future?

I often think of how compliant we have become. Anytime you use Google or have an iPhone or any phone for that matter, you have to ‘agree to the terms’, every Apple update there is something new to comply with. This is a problem of how they suck you into the matrix. Your land line phones never did this.

The transhumanist tech companies know the addicted will comply and agree to any terms to suit their addiction to technology and don’t bother to read anything they want you to bend the knee for. The trade off for entertainment for the soul has been around for a long time. But the manner in which these transhumanist are capturing people should be unsettling to anyone who can think.

x.com/DrJackKruse/st…

2. Wireless radiation is being used to surveil people without their knowledge or consent, even if they aren’t wearing a “smart” device or holding a cellphone, according to the authors of a new study.

The study authors engineering faculty members with the Institute for Systems and Computer Engineering, Technology and Science at the University of Porto, Portugal posted their report Jan. 24, 2025 on the Cornell University open-access research website, arXiv.

The study showcases hardware the authors designed that leveraged ambient wireless radiofrequency (RF) radiation to detect and render a visual image of human activity such as waving a hand or a person’s breathing rate with over 90% accuracy.

Their design involved a thin programmable surface a Reconfigurable Intelligent Surface (RIS) that communicates with computers and artificial intelligence (AI).

3. RIS can manipulate and steer Wi-Fi signals in controlled ways, explained Fariha Husain, manager of Children’s Health Defense’s (CHD) Electromagnetic Radiation (EMR) & Wireless Program. “By adjusting how radio waves reflect off this surface, the researchers were able to enhance signal-sensitivity and improve their ability to detect subtle human movements.”

Husain said:

“RIS panels can be strategically placed to optimize wireless signal reflection and steering and may take any shape or be integrated into objects. Indoors, they can be mounted on walls, ceilings or furniture.

“Outdoors and in urban areas, they can be installed on offices, airports, shopping centers, lampposts and advertising billboards. Additionally, RIS panels will enable smart city surveillance by tracking pedestrian and vehicle movement.”

RIS hasn’t been incorporated into current wireless networks but it’s in the works for 6G, according to W. Scott McCollough, chief litigator for CHD’s EMR & Wireless cases.

“Future 6G networks will have RIS functionality built in,” he said, “and it would not surprise me if they don’t implement RIS in the future 5G updates.”

Read 8 tweets

Oct 11

What do you know about the Stiles-Crawford effect in a healthy eye? What if I told you this effect is how we sharpen central vision and narrow the periphery of the retina from too much blue light. Would you believe it? Did you know melanopsin has a specific topographic map on a healthy retina? A lesson no has taught you is incoming.

2. All opsins are topologic insulators. You might want to read that threadroll above now to understand topology well.

Topology changes in a cell = geometry change of cristae = UPE change from mitochondria = the optical signal changes in the same tissue altering physiology.

So what happens if you sustain mitochondrial damage in your retina’s colony of mitochondria to the Stiles Crawford effect?

What are the implications?

3. The Stiles–Crawford effect (SCE) is the human eye’s phenomenon of reduced light sensitivity when light enters the pupil from its periphery compared to the pupil’s center. This effect is due to the optical properties of photoreceptors, which act as waveguides and are aligned to channel light towards the fovea, the central point of vision. The SCE makes vision less sensitive to light entering the periphery, thus reducing glare and improving visual clarity. It also keeps a lid on the amount of blue light the periphery the retina gets.

This sharpens vision, makes myopia, glaucoma, cataracts, hyperopia, and AMD almost impossible to get. Makes one resistant to mental illness too. Makes one impervious to diabetic transformations. Makes neurodegeneration rare.

You feeling me yet?

Read 13 tweets